Anterior segment phtography shows deposits of amyloid material in pupillary border and anterior lens surface and irregularities of the pupillary border. Amyloid Polyneuropathy (FAP) type I is an hereditary, multissistemic disorder, characterized by extracellular deposition of amyloid material. It is caused by a mutation of the Transthyretin (TTR) gene- val30met. This anterior segment photography of the right eye of a 60 years old woman with Amyloid Polyneuropathy (FAP) type I (18 years after diagnosis and 15 years after liver transplantation), shows deposits of amyloid material in pupillary border and anterior lens surface and irregularities of the pupillary border. She has no no light perception on both eyes and presents with keratoconjunctivitis sicca, secondary glaucoma and multiple amyloid material vitreous opacities.